Clinical Exmination. Muscle weakness, muscle atrophy, myalgia.
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Accession number;02A0539930
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| Title;Clinical Exmination. Muscle weakness, muscle atrophy, myalgia. |
| Author;
INABA AKIRA
(Tokyoishidai I Byoin Kensabu)
MIZUSAWA HIDEHIRO
(Tokyoishidai Daigakuin Noshinkeikinobyotaigaku)
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Journal Title;Clinic All-round
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Journal Code:Z0697A
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ISSN:0371-1900
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VOL.51;NO.;PAGE.1094-1098(2002)
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| Figure&Table&Reference;TBL.1, REF.3 |
| Pub. Country;Japan |
| Language;Japanese |
| Abstract;The muscle weakness can be caused by the disorder in any position in the route from the cerebrum primary motion area to the muscle. The muscle atrophy appears by the lesion of secondary motoneuron and skeletal muscle, and it does not occur in principle in the primary motoneuron failure if there is no effect of invalidness. The muscular pain occurs, when inflammation and damage are generated in the muscle and after the intense motion is carried out. The needle electromyogram can be made easily if it is carried out in the muscle with lower muscle force. There are formation of the perfect interfacial waveform, muscle action potential of short continuance and low amplitude, and early mobilization as myogenicity changes. In the muscle biopsy, the following are carried out usually as a histochemical dyeing using the freezen sample : Hematoxylin eosin (HE) dyeing, ATPase dyeing, Gomori trichrome modification, NADH-TR dyeing. |
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