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Accession number;04A0178224
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| Title;A Case of Uveal Effusion Syndrome Following Laser Iridotomy |
| Author;
MATSUMOTO YUKIHIRO
(Dokkyo Univ. Sch. of Med., Koshigaya Hosp.)
CHIKUDA MAKOTO
(Dokkyo Univ. Sch. of Med., Koshigaya Hosp.)
UCHINO YASUSHI
(Dokkyo Univ. Sch. of Med., Koshigaya Hosp.)
OMATA HITOSHI
(Dokkyo Univ. Sch. of Med., Koshigaya Hosp.)
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Journal Title;Journal of the Eye
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Journal Code:Y0754A
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ISSN:0910-1810
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VOL.21;NO.2;PAGE.265-269(2004)
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| Figure&Table&Reference;FIG.6, REF.23 |
| Pub. Country;Japan |
| Language;Japanese |
| Abstract;We describe a patient who underwent laser iridotomy (LI) for acute primary angle-closure glaucoma (PACG) and subsequently developed uveal effusion syndrome (UE) without nanophthalmos. The patient, a 67-year-old male, was diagnosed with PACG by a local ophthalmologist. Intraocular pressure (IOP) was 56 mmHg and the angle appeared closed. Treatment with latanoprost, pilocarpine, oral acetazolamide and i.v. hyperosmotic agent did not lower IOP; LI using green laser then lowered IOP to 16 mmHg. Three days later, fundus examination revealed choroidal detachment (CD); notable CDs then developed in all quadrants and retinal detachment associated with 180 degree inferiorly. The leopard-spot pattern characteristic of UE appeared on fluorescein angiography. A-scan ultrasonography disclosed no nanophthalmos; computerized tomography revealed a normal-sized eye with abnormally thickened sclera. Oral and i.v. steroid therapy were tried before proceeding to surgery. The UE resolved after 2 months. This was a case of idiopathic UE with abnormally thickened sclera, suggesting that LI together with latanoprost may induce UE. (author abst.) |
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