Visceral hypocirculation as an early manifestation of cardiac rupture caused by myocardial infarction

Accession number;06A0557946
Title;Visceral hypocirculation as an early manifestation of cardiac rupture caused by myocardial infarction
Author; SAKAGAMI JUNICHI (Kyoto Prefectural Univ. Medicine) KATAOKA KEISHO (Kyoto Prefectural Univ. Medicine) SHINODA MISUMI (Kyoto Prefectural Univ. Medicine) TANIGUCHI HIROYA (Kyoto Prefectural Univ. Medicine) BABA TAKEHIKO (Kyoto Prefectural Univ. Medicine) TOSA MASATOSHI (Kyoto Prefectural Univ. Medicine) TAII ATSUKO (Kyoto Prefectural Univ. Medicine) MOTOYOSHI TOMOKO (Kyoto Prefectural Univ. Medicine) ITO REIKO (Kyoto Prefectural Univ. Medicine) YASUDA HIROAKI (Kyoto Prefectural Univ. Medicine) YAMAJI TAKESHI (Setsunan General Hospital) MITSUFUJI SHOJI (Kyoto Prefectural Univ. Medicine) OKANOUE TAKESHI (Kyoto Prefectural Univ. Medicine)
Journal Title;Med Postgrad
Journal Code:X0599A
ISSN:0285-4716
VOL.44;NO.3;PAGE.255-259(2006)
Figure&Table&Reference;FIG.7, REF.9
Pub. Country;Japan
Language;English
Abstract;A 64-year-old female was rushed to the emergency unit by ambulance and admitted to the hospital under the diagnosis of cardiac tamponade. Pericardiocentesis revealed hemopericardium. Emergency coronary angiography showed the disruption of the proximal left circumflex branch. Although the systemic blood pressure was normotensive, abdominal duplex Doppler ultrasonography demonstrated markedly lowered blood flow in each of the celiac trunk, superior mesenteric artery, proper hepatic artery, splenic vein, and portal vein. This is the first report in which visceral perfusion had decreased in the early stage of cardiac rupture following myocardial infarction. We should be careful about nonocclusive mesenteric ischemia in the event of myocardial infarction, even if the systemic pressure remains stable. (author abst.)